Strangles in Horses

Hi Readers,

So after a recent outbreak of Strangles very close to home, I thought I would research this disease in greater detail. Having been aware of the disease for many years now, the necessity of knowing its whereabouts is crucial when caring for my horse.

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My biggest fear when hearing about a recent Strangles case is always how contagious it is. As a young child I was always told not to go on public bridle paths when the yard nearby has a case of strangles. However, Strangles is not actually airborne unlike equine flu so I am not sure how effective this piece of advice actually was.

It is a respiratory infection caused by Strep. Equi, which causes depression, loss of appetite/difficulty eating, raised temperature, cough, nasal discharge, swollen glands in throat and rupture of glands with pus visible as symptoms. The disease can be diagnosed by a blood test; some issues with detecting Strangles include its tendency to be mistaken by the common cold or allergies. In severe cases, Strangles can be fatal in 1% of cases when abscesses develop in other body organs which grow and rupture, a form known as ‘bastard strangles’. Another life threatening complication is “Purpura hemorrhagica”. This is widespread small bleeding along with fluid accumulation of the limbs, eyelids and gums. The outer accumulation of fluid can be so extreme that circulatory failure and death can occur.

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From seminars I have attended in farm animal health, the concepts are near enough always the same. If you go out of your way, speaking to the yard owner and educating people on the yard, to prevent the disease entering and infecting your animals in the first place, the hard decisions that come when the disease gets very severe will not have to be made. Prevention is always better that a cure! To try to prevent the disease from infecting an animal a number of precautions can be taken:

  • Avoid contact/sharing tack or equipment with horses whose health status you do not know (e.g at shows)
  • Don’t overcrowd a yard – diseases spread more easily
  • Quarantine horses that come on the yard, care should be taken with who comes into contact with the new arrival and what other animals they have to attend to  
  • If people have arrived from an affected yard, their movement should be restricted
  • Horses should be up to date with their vaccinations

There is also a vaccine to prevent Strangles. The Equilis StrepE is a vaccine that contains the active substance live deletion mutant Streptococcus equi bacteria. This is the first vaccine to be licensed for horses in the EU against strangles. All members should follow a vaccination schedule on a yard to be sure this vaccine is effective and to prevent the closure of a yard.

I would be really interested in any experience you have had with the disease. Feel free to comment below!

Sol

References

http://www.equine-strangles.co.uk/disease-control.aspx

http://www.theodora.com/drugs/eu/equilis_strepe_veterinary.html

http://www.equine-strangles.co.uk/about-strangles.aspx

 

 

 

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Foot Rot in Sheep

Hi Readers,

Firstly, apologies for not updating my site recently; as I’m sure most of you are aware it’s lambing season, so I am currently balancing studying and lambing shifts! One of the things I’ve managed to do a lot of this season is foot trimming the ewes before we turn them out with their lambs. I’ve realised how much physical strength is required in order to turn the ewes over – a couple of bruises later, and I think I’m getting the hang of it!

What I wanted to look into on this post, is the reason I’ve found some ewes with bad feet who have clearly suffered from foot rot. This means I’ve injected quite a few ewes intramuscular with Alamycin – another skill I’m quickly getting the hang of!

Scald and footrot are caused by the bacterium “Dichelobacter nodosus”. This is contagious and can be passed onto other sheep especially in the UK climate – very damp and perfect temperature. It causes 90% of lameness in sheep in the UK. It may also be on set during lambing time, when the ewes are in for lambing and the straw becomes wet and warm.

The interdigital skin in the feet becomes red and swollen and covered by a thin layer of white exudate (a mass of cells and fluid that has seeped out of blood vessels or an organ, especially in inflammation). I treated any ewes that I found with this condition using blue spray, alamycin if very bad (to the point where there was a very strong smell) and leaving it untrimmed. We will move these ewes into a separate paddock to reduce risk of infection to other sheep and so they can be treated again in a few weeks. When I was trimming feet during pre lambing checks we used a foot bath which also proved very effective in helping to heal the interdigital dermatitis.

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Sol

References

http://www.nadis.org.uk/bulletins/lameness-control-in-sheep.aspx

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Ethics on the topic of dissections in schools

Hi Readers,

After doing a number of dissections this week while studying the respiratory system of different species, one of the questions I needed to answer for a core practical was “Describe the approaches that were taken to ensure the ethically responsible use of animals in this practical activity”.

This got me thinking on what the different opinions were on dissections from an animals-rights activist’s point of view as well as those in favour of the dissections.

When doing some research I found a PETA article called “Dissections: Lessons on Cruelty”. It talks about the different animals that are commonly dissected, such as frogs and mice. It also mentions the methods in how the animals are obtained for dissection, implying that escaped cats, for example, were in fact stolen from their homes by suppliers of dissection animals. The article claims “Classroom dissection desensitizes students to the sanctity of life. Research has shown that a significant number of students at every educational level are uncomfortable with the use of animals in dissection and experimentation.” After seeing some of my peers uncomfortable with the dissection, I spoke to one of them about what exactly made them uncomfortable when watching/carrying out these practicals especially the compulsory core ones. She said: “I think sometimes it’s wasting animals. The core practical we did was dissecting a locust, which I was more comfortable with as insects are annoying. Also, if the animal is already dead then I don’t mind as much, for example, if it comes from a butcher’s. If it was a full cow on the other hand, I wouldn’t be okay with killing it just for dissecting. It could be used to feed a lot of people so dissecting it is just a waste. If a laboratory had to kill a healthy cow to do research I’d understand that but not for a school dissection as this is just to see what’s inside. I don’t really like the smell during these practical activities either, or the sight of blood.” Another peer commented that he was “just afraid of rats” so didn’t want to watch the dissection I was chosen to do in front of the class.

After reading another article, I read a statement that I found very concerning. Heidi Blake writes for the telegraph (2010) “Schools are abandoning the practice of cutting up frogs, rats and animal organs which has been a mainstay of biology lessons for generations, out of concern for squeamish pupils and fears that they could turn their scalpels on each other.” The article was based on the reduction of dissections taking place now due to health and safety concerns and that most of these practicals are now being replaced by videos or the teacher’s are carrying out demonstrations. The violent element of this statement surprised me as only a few generations before my own would claim that they remember hating biology due to having to capture their own and then dissecting the animal (mostly frogs). It leaves me with a few questions: is there more crime occurring now? Is it more between young people? Do we just hear about incidents more because of social media/ the news on national and international television?

In my opinion, I think by commercially killing animals to be eaten we are doing just as much harm as if we killed them for these practicals. We are claiming it is fine to kill them in order to fulfill a basic human need, but not fulfill the need of potential future scientists – this practice may influence a person to follow this career path who could go on to cure a human disease. This forms a cycle – treatment from disease is also a basic human need. In my experience, I have enjoyed dissections and recall wanting to dissect any animal as soon as I moved up to secondary school in order to learn more about how my own body functions as well as that of different animals. I have learned valuable skills such as accuracy, patience and steadiness. I also think diagrams of these animals usually don’t help at all and oversimplify structures; in a locust for example the spiracles are not obvious dots and if I hadn’t done the dissection myself I wouldn’t have understood fully the structure of the chitin supporting the tracheae, having not seen it under the microscope.

I have seen that many A level students are also currently carrying out these practices and would be very interested in hearing your views.

Sol

 

References

http://www.peta.org/issues/animals-used-for-experimentation/animals-used-experimentation-factsheets/dissection-lessons-cruelty/

http://www.telegraph.co.uk/education/educationnews/7668210/Schools-abandon-dissection-in-Biology-lessons-over-health-and-safety-fears.html

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Respiratory Systems – Chickens vs. Sheep

Hi Readers,

This week I thought I’d investigate different respiratory systems, and the problems that can occur with them, in two different species in particular after a topic I covered in biology. I wanted to see the differences between mammals and birds.

Sheep

All mammals have the same respiratory system but adapted to their size and shape. The equation for respiration is:

oxygen + glucose -> carbon dioxide + water (+ATP)

Gas Exchange

Diffusion is when molecules move from an area of high concentration to an area of low concentration. The experiment below shows a container separated with a semipermeable membrane, dividing an area with a high concentration of a molecule (red dots) from an area with lower concentration. The membrane allows the molecules to move from one side to the other. Over time, the molecules will move from the area of high concentration to the area of low concentration. They do this until the overall net movement is equal. It reaches equilibrium.

Screen Shot 2017-01-28 at 07.42.48

This happens during respiration – oxygen and carbon dioxide are often highly concentrated on opposite sides of a cell membrane. Diffusion allows gas exchange to occurIn animals with a closed circulatory system (both chickens and sheep) – gas exchange takes place across capillaries.

Unlike humans who have two lungs – the left divided into two and the right into three lobes, sheep have highly segmented lungs consisting of two lobes in the left lung and four lobes in the right lung with the bronchus of the right cranial lobe coming directly from the trachea.

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Sheep airways are similar to humans’ in terms of where epithelial cell and mast cells (type of white blood cell) are found. The sheep lungs have a significant population of pulmonary intravascular macrophages that are important in engulfing foreign particles and pathogens.

Inside the nasal cavity of mammals are ciliated epithelial cells, which are used to waft any bacteria and unwanted foreign bodies down the throat to the stomach. The stomach acid and digestive enzymes then kills them. There are also goblet cells, which are used to produce mucus. This catches the unwanted microbes; the moist and warm conditions make it even stickier to ensure the microbes do not enter and grow in the body.

Sheep are obligate nasal breathers, meaning they mostly breathe through their nose but do possess the ability to occasionally breathe through their mouths. It has been suggested that obligate nasal breathing is an adaptation very useful in prey species, as it allows an animal to keep eating, especially as sheep are grazing animals so preserve their ability to detect predators by scent. Humans however, breathe via either mouth or nose. The advantage of breathing through the nose is there is less chance of an infection as there are no ciliated epithelial cells or goblet cells, making mucus, in the mouth for protection.

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In mammals, the trachea splits off into two bronchi, which then break off further to form bronchioles. The bronchioles have alveoli attached to the end of them which is where gas exchange occurs. There are many adaptations of the alveoli that allow it to exchange gases efficiently. These include:

  • Lining of the lung and capillaries only one cell thick – so gases only have to diffuse over a short distance
  • Large surface area – lots of oxygen can diffuse into blood stream at once
  • Good blood supply – oxygen is carried away to the cells as soon as it has diffused
  • Liquid surfactant – gases must diffuse in liquid to be able to diffuse into the blood
  • Phagocytic white blood cells – engulfs bacteria to help protect the body
  • High concentration gradient – gases can diffuse in and out of the alveoli quickly due to the concentration gradient (blood has high concentration of CO2 produced by respiration but not much O2; lungs have high concentration of O2 but not much CO2

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Ventilation movements also maintain the concentration gradients because air is regularly moving in and out of the lungs.

Inspiration:

  • The diaphragm muscles contract and the diaphragm flattens and moves down
  • The intercostal muscles contract, which makes the ribs move up and out
  • The volume of the thorax increases. As the volume increases the pressure decreases
  • Air flows in down a pressure gradient
  • Oxygen diffuses into alveoli along the concentration gradient. In the alveoli, oxygen dissolves in liquid, which then diffuses the short distance into the blood capillaries

Expiration:

  • The diaphragm muscles and the intercostal muscles relax
  • Lungs return to normal size (recoils)
  • Ribs move in and down and the diaphragm moves upwards
  • Volume of the thorax decreases and the pressure increases
  • Air moves out of the lungs and out of the body down a pressure gradient

Chickens

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The openings to the nasal cavity (nares) are on the top beak. Within the cavity are:

  • Squamous epithelium – a single layer of flattened cells
  • Ciliated epithelium – cube shaped cells with cilia or hairs that trap foreign bodies (dust/bacteria)
  • Olfactory membrane – gives the sense of smell

The epithelium of the nasal cavity is covered in mucosal glands, which produce mucus that helps to keep foreign material from entering the body through the respiratory system.

Oropharynx (mouth and pharynx)

The oropharynx consists of the mouth and the pharynx (the cavity behind the nose and mouth, connecting them to the oesophagus). Behind the base of the tongue is the rima glottidis  (opening into the larynx). There are no vocal cords, epiglottis or thyroid cartilages that are normally found in mammals.

Trachea

Held open permanently by 108 to 125 cartilaginous rings. The trachea is lined with mucociliary epithelium where the hair-like cilia move foreign materials, such as dust, up and out of the trachea. Numerous mucous secreting glands are also found in the tracheal lining.

Bronchi

The trachea divides at the syrinx (found at the end of the trachea) into the left and right bronchi. The cartilaginous rings of the bronchi extend from the syrinx to where the bronchi enter the lungs. Ciliated epithelium with numerous mucous glands lines the bronchi. Some are lined with squamous epithelium.

Gas exchange

Leading off from the bronchi in the lungs are a large number of extremely small air capillaries that are interlocked with the capillaries of the lung circulatory system. These interlocked capillaries are the lungs’ gas exchange system and are very thin. The cell layers that separate the two systems (the blood circulatory system and the air supply system of the lungs) are:

  • Single cell epithelial wall of the air capillary
  • Base membrane one cell thick
  • Single cell epithelial wall of the blood capillary

Lungs and Air Sacs

Fowls do not actually have a diaphragm unlike the respiratory system in mammals. The chicken lung is also much denser than a mammals’. Unlike other species, the avian lung has very little elasticity.

The air sacs are very thin walled coming off of the bronchi (NOT FOUND IN THE LUNGS LIKE MAMMALS!). Some of these sacs also connect to many of the larger long bones to form the pneumatic bones. These make the bones lighter and are an advantage for flight.

It is very difficult to see the air sacs in a bird because they are so thin and transparent. It is believed the walls are lined with simple squamous epithelium although there is some ciliated epithelium in some areas. There are very few blood vessels present which indicates that these sacs play no part in gas exchange. The air sacs provide a very high volume of air for use by the lungs on inspiration and expiration. Therefore the lungs, with help from the air sac system, supply the oxygen necessary to fly. The air sac system permits the bird to change its center of gravity, which also aids flight.

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Inspiration, Expiration and Movement of the Body Wall

Respiration is the result of movement of the body external wall that either causes an increase or decrease in the volume of the cavity – this is because, as I mentioned earlier, fowls do not have a diaphragm. These changes in volume cause changes in pressure inside of the lungs and this causes air to either enter or leave the system.

Inspiration

  • Ribs are drawn forward
  • Sternum pulled downwards
  • Volume increases
  • Pressure decreases
  • Size of the cavity increases crosswise and from front to back

The increase in size and volume lowers the pressure in the lungs and air sacs and results in air from outside being drawn in to equalise the pressure. Inspiration occurs as the air moves into the lungs and air sacs.

Expiration

  • Muscles contract
  • Ribs and sternum return to their original position
  • Volume of the cavity reduces
  • Air pressure in the system increases

This decrease in size and volume increases the pressure in the lungs and air sacs and results in air from inside being pushed out to equalise the pressure. Expiration occurs as the air moves out of the lungs and air sacs.

Chickens actually require two respiratory cycles!

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(a flow diagram I made to try to understand how air travels through the respiratory system)

Similarities Differences
Both have air sacs Chicken’s air sacs are not inside the lungs, a sheep’s are
Air is drawn in and forced out of the lung due to change in pressure – by either movement of the body wall or by contraction of intercostal muscles There are very few blood vessels in air sacs so probably aren’t used in gas exchange like in sheep
They are both obligate nasal breathers but can breathe through their mouths and tend to do so when stressed There are no vocal cords, epiglottis or thyroid cartilages in a chicken like those that are found in sheep
Both produce mucus to catch bacteria and other foreign bodies – for protection Chickens don’t have a diaphragm, sheep do
Both have single cell layers in capillaries to allow for quick diffusion Chickens have very little elasticity in lungs like in sheep
Both have tracheas lined with epithelial cells A chicken lung is also much denser than a sheep’s
Both have closed circulatory systems Respiration in birds requires two respiratory cycles to move the air through the entire respiratory system. In mammals, only one respiratory cycle is necessary
When comparing birds and mammals of similar weight, birds have a slower respiratory rate
The respiratory system of birds is more efficient than that of mammals, transferring more oxygen with each breath
Holding a bird “too tight” can easily cause the bird to suffocate, with sheep this is harder to do


Respiratory Diseases in Sheep and Chickens

Sheep

Name Description Image Symptoms Treatment
Pasteurellosis  Causes septicaemia in young lambs, pneumonia in older sheep, and mastitis in ewes. It is a respiratory disease caused by M. haemolytica  Screen Shot 2017-01-27 at 20.18.10 Depression, lethargy, inappetance, increased respiratory rate, fever Oxytetracycline is the antibiotic used for pasteurellosis as there are few antibiotic resistant strains in sheep
Parasitic Chronic suppurative (pus formation) pneumonia/lung abscesses Very common in mature rams but are difficult to identify by inspection alone  Screen Shot 2017-01-27 at 20.45.05 Weight loss although appetite may appear normal, rectal temperature is often slightly elevated (up to 40.0°), higher respiratory rate, nasal discharge Penicillin is the antibiotic used for chronic respiratory disease 
Ovine pulmonary adenocarcinoma OPA is a contagious, viral, neoplastic disease of the lungs of sheep   Chronic weight loss, dyspnea, crackles, and copious amounts of serous nasal discharge from accumulated lung fluid in an adult sheep No specific treatment is available, affected sheep must be culled

 Chickens

Name Description Image  Symptoms Treatment
Gapeworm Caused by the worm “Syngamus trachea” -the adult worms live in the chicken’s tracheaChickens acquire the disease by eating infected earthworms/snails    Coughing, head-     shaking , ‘gaping’ mouth appearance Wormer such as Flubenvet or Solubenol
Infectious Laryngotracheitis   Herpesvirus causes significant inflammation of the upper airways, and can have a high mortality in chicken flocks   Gasping, coughing of mucus and blood, drop in egg production,sinusitis No definitive treatment (can only try to manage any secondary problems, such as secondary bacterial infections)
Infectious Bronchitis  Caused by a highly infectious virus (coronavirus)  Mortality rate is higher in young birdsThere are vaccines available which are given to chicks   Depression, gasping, coughing, tracheal rales (crackling), nasal discharge, drop in egg production/ producing soft-shelled eggsWhen the kidneys are affected – increased water intake and scouring No treatment (but if a secondary bacterial infection is suspected then antibiotics may help)

I found the similarities and differences between the two animals very interesting and will find out more about different animal families and their respiratory systems. The diseases were also interesting to so I could learn more about why each species is more susceptible to certain diseases than the other. If anyone has seen any respiratory diseases perhaps on work experience or out practising, I would love to know more about what you saw and how it was resolved! I once saw a horse with Chronic Obstructive Pulmonary Disease (COPD)!

Sol

References

http://www.shmoop.com/animal-movement/animal-respiration.html

http://jtd.amegroups.com/article/view/3377/html

http://allergenix.com.au/wp-content/uploads/2013/08/SheepModels.pdf

https://en.wikipedia.org/wiki/Obligate_nasal_breathing

http://www.poultryhub.org/physiology/body-systems/respiratory-system-thermoregulation/

http://www.s-cool.co.uk/a-level/biology/gas-exchange/revise-it/gas-exchange-in-humans

http://www.examstutor.com/biology/resources/studyroom/organs_and_systems/gas_exchange_in_mammals/ventilation_mammals.php?style=printable&PageTitleFull=2%20Ventilation%20in%20mammals

http://www.ehow.com/info_12289822_chickens-breathe-through-mouths.html

http://www.flytesofancy.co.uk/chickenhouses/Worming_Poultry.html

http://www.evolutionfarmvets.co.uk/respiratory-disease-chickens

http://www.thepoultrysite.com/diseaseinfo/83/infectious-laryngotracheitis-ilt/

http://www.infectious-bronchitis.com/signs-lesions-ib.asp

http://www.poultrymed.com/Poultrymed/Templates/showpage.asp?DBID=1&LNGID=1&TMID=103&FID=1519

http://www.nadis.org.uk/bulletins/respiratory-disease-in-adult-and-yearling-sheep.aspx?altTemplate=PDF

http://www.peteducation.com/article.cfm?c=15+1829&aid=2721

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Marine Mammals – Killer Whales

Hi Readers,

After hearing the news that SeaWorld’s orca ‘Tilikum’, the whale that killed trainer Dawn Brancheau, has died I thought I’d blog about the controversial topic of keeping the orca’s in captivity. Having recently applied for work experience at an aquarium, working with sea mammals, I thought the topic would be very relevant.

I suppose the arguments for keeping these killer whales in captivity meant the animals could provide a rare opportunity to do crucial research, and the breeding program at SeaWorld does help with increasing killer whale numbers. The killer whale’s only predator is mankind. Although Orcas are not an endangered species, some local populations are considered threatened or endangered due to pollution, depletion of prey species, conflicts with fishing activities and vessels, habitat loss, and whaling.

However, “If SeaWorld didn’t exist, would our understanding of wild killer whales be significantly reduced? I think the answer to that is no, it would not,” says a veteran marine-mammal researcher who works at the National Oceanic and Atmospheric Administration. Furthermore, most animal right activists have said that releasing the orca’s into the wild now that SeaWorld has put an end to its breeding program would not be a wise idea. Most of the killer whales at SeaWorld have live there all their lives and would not survive without human help.

In my opinion, I think breeding programs to increase numbers of these beautiful creatures would be very beneficial, especially as it is our fault for reducing numbers – by whaling or oil spill contamination to their habitats. However, I do agree that the public should get to see these wonderful animals, especially if the money they use to buy a ticket can go towards breeding more of these magnificent creatures. On the other hand, getting them to do tricks and making them act outside of their normal behaviour, perhaps isn’t the way to do it. Unlike the orcas in the ocean, the killer whales in captivity need antibiotics, antifungals, and even antidepressants to maintain their health and well-being. I strongly disagree that this is okay…I believe any animals mental health is as important as ours.

A possibility for these animals could be moving to sea pens, this would allow the orca’s to be in a more natural habitat.

The Keiko example

 

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The killer whale featured in the 1993 film “Free Willy” is often cited in the debate over sea pens.

Keiko was captured off the coast of Iceland in 1979 and trained to perform at theme parks. Many years later, the orca was transported to a sea pen in Iceland in 1998 after spending several years at a Mexico City theme park. Keiko swam away during a short cavort outside of the pen while accompanied by caretakers on a ship. He later turned up in a deep inlet in Norway and was found playing with children and fishermen. The whale died a few months later of acute pneumonia.

SeaWorld trainers have said the experience showed that sea pens were not a safe environment for orcas. Others countered this view, saying the experience with Keiko taught experts how to build a better sea pen. If better pens can be made that will not allow the whales to get pneumonia, this case study proves that orca’s could survive if transferred from theme parks to sea pens.

I’d love to hear other people’s arguments for and against the captivity of any marine mammal, as well as the orca. Feel free to leave a reply!

Sol

References

http://www.takepart.com/feature/2016/06/14/killer-whales-new-life-after-seaworld

http://www.motherjones.com/environment/2014/12/seaworld-killer-whale-orca-science-blackfish

http://www.seaworldofhurt.com/features/seaworld-trainer-dawn-brancheau-death/

http://www.killer-whale.org/killer-whales-endangered/

http://wwf.panda.org/about_our_earth/teacher_resources/best_place_species/current_top_10/orca.cfm

http://www.latimes.com/business/la-fi-seaworld-sea-pens-20160317-htmlstory.html

http://www.bbc.co.uk/news/world-us-canada-38531967

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Bloat – Canine

Hello Readers,

After spending a week on work experience at a kennels one of the things I found was the importance of a dog’s diet as it could lead to complications with the animals health. Bloat was one of the problems I was taught was common in dogs if the diet was not kept consistent. I did some research into ‘bloat’ in dogs or medically known as Gastric Torsion.

I found that this syndrome is caused by the stomach filling with gas and causing it to twist. This can be caused by anxiety as commonly seen in kennelled dogs which I assume is why it was brought to my attention, as the dogs become stressed in a new and unfamiliar environment.

If changed onto very fermentable food, the dog may have gas build up in the stomach. Excessive eating and then vigorous exercise afterwards may also be a factor to consider which is why it was important I knew the correct amount of food needed to for each breed and size of dog; especially as it is most commonly seen in large, deep-chested breeds.

The following symptoms may indicate a case of bloat:

  1. Anxiety – Pacing around or trying to vomit, without success
  2. Too much air intake
  3. Dribble or saliva from dog’s mouth
  4. Gut bloating – a distended (swollen) stomach seen

Like in humans if we can’t breathe we begin to panic, the expanded and twisted stomach pushes the diaphragm and restricts breathing making the situation much worse. While the stomach is twisted, changes occur in blood levels of oxygen leading to the death of cells in other organs as well as in the stomach. It must see a vet immediately as this syndrome is fatal.

bloatpic

Have any of you had a dog with a case of bloat/seen it in a practice?

Sol

 

References

http://www.k9magazine.com/bloat-in-dogs/

http://www.thekennelclub.org.uk/health/for-owners/dog-bloat/

http://lapoflove.blogspot.co.uk/2012_05_01_archive.html

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Highly Pathogenic Avian Influenza H5N8 in Europe

Hello Readers,

As most of you I’m sure are aware, there has been a new strain of avian influenza infecting birds in Europe and has recently moved to Wales. I composed some questions that I wanted responses to and did some research into the answers. In the veterinary profession our vets work to ensure human health is taken into account when working towards good animal health.

Where has this originated from?

According to ‘Reuters’ and ‘Fox News’ the first case of the H5N8 strain of avian flu was detected in Denmark on a poultry farm. About one-third of 30 ducks at a farm north of Copenhagen were killed by the same virus that had been found in Denmark in wild birds.

http://uk.reuters.com/article/us-health-birdflu-denmark-idUKKBN13G0WI

How is it spread between birds?

Infected birds can shed avian influenza A viruses in their saliva, nasal secretions, and feces. Susceptible birds become infected when they have contact with the virus as it is shed by infected birds. They also can become infected through contact with surfaces that are contaminated with virus from infected birds. Domesticated birds (chickens, turkeys, etc.) may become infected with avian influenza A viruses through direct contact with infected waterfowl or other infected poultry.

https://www.cdc.gov/flu/avianflu/avian-in-birds.htm

How are these strains changing so quickly?

As well as the mutations we are familiar with such as bases being substituted, there is another type of mutation; viruses exchange information with one another. These mutations can occur when different kinds of viruses come into contact with one another in a single host. Hypothetically, for example, a virus that is easily spread from person to person could exchange genetic information with a highly pathogenic strain of avian flu virus, creating a new strain that can be transmitted easily among humans.

The potential for this type of mutation to occur is greatest when there are many opportunities for the virus to multiply—in large flocks with many infected birds. The virus can spread more quickly in crowded conditions, and birds in high-density flocks may be more susceptible to the disease because the stressful conditions may weaken their immune systems.

Viruses have more opportunities to exchange information where these flocks are in close contact with humans or other domestic animals, raising the potential for a human or a pig, for example, to serve as a host for two flu viruses that can exchange genetic information and become more harmful to humans.

http://www.birds.cornell.edu/birdflu/what-you-should-know/q-a/q-a-10 

Will this put human lives at risk?

WHO write “Human infection with the H5N8 virus cannot be excluded, although the likelihood is low, based on the limited information obtained to date. It should be noted that human infection with H5N6 of related clade 2.3.4.4 has already occurred. WHO will re-assess the risk associated with the virus when more information is available.”

In the past:

  • Humans can be infected with avian and other zoonotic influenza viruses, such as avian influenza virus subtypes A(H5N1), A(H7N9), and A(H9N2)
  • Human infections are primarily acquired through direct contact with infected animals or contaminated environments
  • Avian infections in humans may cause disease ranging from mild conjunctivitis to severe pneumonia and even death.
  • The majority of human cases of A(H5N1) and A(H7N9) infection have been associated with direct or indirect contact with infected live or dead poultry. Controlling the disease in the animal source is critical to decrease risk to humans.

http://www.who.int/mediacentre/factsheets/avian_influenza/en/ 

http://www.who.int/influenza/human_animal_interface/avian_influenza/riskassessment_AH5N8_201611/en/

Why are people so concerned if the disease infects domesticated birds?

  • the potential for low pathogenic viruses to evolve into highly pathogenic viruses (we do know that this virus, is in fact, highly pathogenic)
  • the potential for rapid spread and significant illness and death among poultry during outbreaks of highly pathogenic avian influenza
  • the economic impact and trade restrictions from a highly pathogenic avian influenza outbreak
  • the possibility that avian influenza A viruses could be transmitted to humans

http://www.who.int/influenza/human_animal_interface/avian_influenza/riskassessment_AH5N8_201611/en/

Sol

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Pre Lambing Checks – Heptavac P Plus vs. Ovivac P Plus

Hello Readers,

So i thought I’d start off with a few older photos. These photos where taking during lambing at the beginning of this year (2016). I was helping out with herding and loading the ewes to be brought back to the farm, ready to be checked and in for lambing. I was taught how to inject the ewes subcutaneously (I did about 100 in one day).

This injection was ‘Heptavac P Plus’. After some research into this injection tonight I came across a forum discussing the difference between ‘Heptavac P Plus’ and ‘Ovivac P Plus’. I started to look into the difference between the two and how they can prevent pasteurella pneumonia (organism in tonsils – disease released when animal is stressed) and clostridial disease (organisms for this found in soil).

The Heptavac P Plus contains antigens from seven clostridial species. This provides immunity to the lambs via colostrum during lambing. This vaccination is given during pre lambing checks as it must be injected with enough time to make its way into the milk. I also found that this injection is the most expensive of the two.

The Ovivac P Plus doesn’t contain the antigens from as many clostridial species (only 4). It is usually used for store lambs as they need the extra immunity they are not receiving from a ewe’s milk. This boost of immunity increases chance of survival, especially in the winter months when there is a peak in number of cases of these diseases.

I rang the farmer I did this with back in January/ February time, who I have worked with throughout the year. He told me he sold some store lambs a few years ago and knew that the customer injected the lambs with Ovivac P Plus as a little bit of extra immunity as it moved onto a new farm. The Ovivac is cheaper so more economical to use, if just a precaution.

If any lambs from lambing/ store lambs are kept for replacement they are boosted with the Ovivac P Plus at the time of pre tupping. After this they join the regular routine of the older ewes, receiving the Heptavac P Plus pre lambing.

This particular farmer chooses to use the Heptavac P Plus on all 500-600 ewes as the ewes are in constant contact with each other in sheds during lambing so does not want these diseases spreading through and killing his flock as it is the biggest killer of sheep – despite the large cost.

I wonder if any readers would like to share their experience with the two; what routine do you use? Is the Heptavac P Plus worth the extra money? Does anyone give Ovivac P Plus to ewes as they are in a low risk zone perhaps? I would love to hear your thoughts.

injectionphotoinjectionphoto2

The Forum can be found at :

https://thefarmingforum.co.uk/index.php?threads/ovivac-p-plus-or-heptavac-p-plus.11973/

References :

http://www.hodgesandmoss.co.uk/sheepvaccines.html

Sol

 

 

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Welcome

Hello all,

After attending a Vetmed Link course this weekend I became very interested in sharing my journey through work experience, voluntary work and reading up on current affairs in the veterinary profession. I will write about my findings on work experience; not only what I practically learned but I also hope to comment on any ethical implications I came across – any photos will be placed here as well. I will also try to discuss topics that are in the news, in the veterinary world.

 

Sol

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